It always takes a couple of years for data to start filtering through, but in 2018, a survey of veterinary professionals believed that rates of obesity are increasing year on year. It is thought that 51% of dogs and 44% of cats in the UK are overweight. That is just over half of the population of dogs and nearly half of the population of cats. These numbers are up on the last data sourced in 2015. 100% of veterinary professionals are concerned about the prevalence of obesity and the health issues it can lead to. In the words of James Smith PT, we’d love to tell you it’s as simple as a calorie deficit, but here at My Pet Nutritionist, we know that obesity is in fact a multifactorial condition, which can make it a hard nut to crack. But we also know that the health of your pet depends on it. So, we’re not about to grab a hammer, instead we’re going to take a look around the cookware store and find the right nutcracker to do the job. Fancy a trip? The Health Issues Associated with Obesity: Being overweight increases the risk factors for developing: Cancer Diabetes Heart disease Osteoarthritis and degeneration of joints Urinary bladder stones Surgery complications Respiratory difficulties Kidney disease All things considered, its easy to see why obesity significantly reduces lifespan and why it is something that really needs to be addressed. What is obesity? Obesity is defined as an accumulation of excessive amounts of adipose tissue. It is generally a state of positive energy balance. When food is ingested, it is digested and metabolised. The body uses the nutrients it needs and converts the main macronutrients to energy. All energy in the body is produced by the breakdown of ATP, adenosine triphosphate. ATP is found in all cells in all the body, but as it is a large molecule, not so much can be stored. In nearly every living thing, this energy comes from the metabolism of glucose. There are three relevant energy systems. 1. ATP-P This is where the body uses all the ATP it has stored in its cells. This is the simplest energy production process; and if we were to consider it in human terms, this is the system that your 100m sprint would utilise. 2. Glycolytic System This system runs on glycogen, which is stored glucose. In human terms, this system provides moderate power and moderate duration. Both the ATP-PC and Glycolytic system are anaerobic, meaning they don’t require oxygen to produce ATP. 3. Oxidative Syste This system, as its name suggests does involve the use of oxygen to product ATP. This system cannot produce energy as quickly as the other two, but it can produce it continually and for a longer duration. This system can use stored carbohydrates and fats for fuel. In human terms, this would be the system that the marathon runner would access! So, in short, glucose is used as energy, but unused glucose can be converted to glycogen to be stored in the liver and muscles. If there is still leftover, then it is converted into fatty acids and stored in adipose tissue. This makes evolutionary sense; there would be a reserve during periods of low food availability. However, as you have noticed, fat too can be used as an energy source, and protein can be broken down into amino acids and then converted to glucose in a process known as gluconeogenesis. This is where the calorie in, calorie out idea comes from. An overabundance of any of the three macronutrients, can be converted into fat for later use. And the idea that obesity comes from reduced activity comes from the reasoning that those reserves aren’t actually used. On a simplistic level, this makes absolute sense and for the pet who is starting to look a little more round than usual, a simple reduction to their daily intake or a longer walk, may just tuck that waist in a little more. But there are many more factors at play here. Fat is the New Norm Do you know your pet is overweight? In an 8000-household study, 68% of surveyed owners report thinking their pet is the perfect weight. 67% of surveyed owners did not see obesity as a concern. Interestingly, a recent report from the NHS in the UK identified that 67% of men and 60% women were overweight or obese. 20% of Year 6 children were also classified as obese. Findings here Whilst we acknowledge there are faults in BMI scores, it still highlights a valid point. Sadly, pet obesity has many parallels with human obesity and according to the NHS, fat is the new norm for humans. As we know, we learn from experience and so, if we are consistently exposed to something, it becomes accepted. If we are consistently exposed to round pets, without a waistline, do we accept it? But, our pets need a waistline. They should tuck-in behind their ribs, and underneath. Whilst you shouldn’t be able to see their ribs, you should be able to feel them. They should not look rectangular from any angle, side or above. What treats are you offering your pet? Whilst for us humans, a decadent chocolate pot may be a weekend treat, dog treats have somewhat lost their novelty. Treats can be a useful resource during training, and in some cases, high value are necessary, but for some, treats are used as a way to prevent feelings of guilt when pets are being left home alone. Depending on how many times you leave the house, these can soon add up! 1. Table scrap We’ve all been there, those eyes looking up at you whilst you’re eating. Whilst that cubic inch of cheese only warrants 68 calories of your daily 2500 (call it 3%), for your 9kg Scottie Dog, that’s nearly 20% of their daily calorie allowance. If begging at the table is an issue, it is possible to remove the pets to
Constipation is the infrequent or difficult passage of faeces. As pet owners, we may pay more attention to the size, colour and consistency of bowel movements and not necessarily notice that our companion hasn’t “gone” for a while. The first hint we may notice is straining when out on a walk with our dogs, or our cat spending more time in the litter tray than usual. When they do go, faeces are dry and hard. Constipation is something that we are asked about regularly here at My Pet Nutritionist, so let’s take a look at what actually occurs in the digestive tract in cases of constipation, what can cause it, and some top tips for managing it. The Digestive Syste Unlike us humans, our pets lack salivary enzymes and so the digestion of their food doesn’t really start until it hits the stomach. As food travels down the oesophagus, it goes through a gateway known as the oesophageal sphincter before getting to the stomach. The stomach is like a washing machine, it churns the food and releases detergent-like compounds to break the food down. Food is turned into a smoothie like consistency known as chyme, which then makes its way to the small intestine. The small intestine is covered in tiny,carpet-like projections known as villi which are vital for nutrient absorption. In short, these villi allow the goodness found in the food to get to the rest of the body. But for that to happen, the chyme needs to be broken down further. Here is where the pancreas comes in. The pancreas is like a factory, it produces enzymes that help to digest food, along with hormones that help control how much sugar is in the blood too. The liver also produces bile acids which are secreted into the small intestine from the gallbladder to help with digestion and absorption of fat. Just to make sure digestion is targeted from all angles, the lining of the small intestine also contains enzymes that break down food too! After the small intestine has done its job, the unabsorbed bits (including fibre) move into the large intestine. But it must go through the ileal brake first. This is like a watchdog; if it spots under-digested food coming through, it tells the upper-gut movements to shut down their party. The large intestine also plays a role in nutrient absorption, simply because it houses trillions of microbes that can help digest things that have so far been indigestible. These microbes also produce messenger molecules to the brain, reducing inflammation and signalling satiety amongst other functions. The large intestine also reabsorbs fluid and electrolytes. During this process,the contents in the gut turn from liquid to solid, so the longer the food remains here, the more water is absorbed and therefore the more solid it is. Sloppy poops spend significantly less time here. At the end of the large intestine, the rectum stores and compacts the waste, before the brain receives the signal to release the accumulated waste though the anus. But how does food move through this system? As the intestine looks much like a computer game from the 1980s, we’re pretty sure gravity isn’t completely in charge. Well, it isn’t. The movement of food through the system is largely thanks to a carefully curated orchestra of muscle contractions. On of these programmes is peristalsis, which are wavelike contractions. Earthworms actually use this mechanism to drive their movement. The other is segmented contractions which generally occur in the small intestine. In the large intestine, mass movements propel the chyme/faeces towards the rectum. What is particularly interesting is that the gut can function independently of the brain, so it can do all of this without any conscious thought. Digestion forms part of the parasympathetic nervous system, appropriately dubbed rest and digest. This leads us into our first possible cause of constipation… Stress When the body feels stressed, resources are redistributed to organs and functions that need them. Sadly, the digestive system is not one of them. Stress can result in a temporary halt to digestive function (of course,stress can also result in projectile diarrhoea, but we never said the body was an easy study did we!). Without the nod to continue its work, waste can sit, waiting for the signal to release. Changes in Routine The digestive system also displays circadian rhythm variations. There is evidence that peristalsis has its own routine. Gastric emptying rates are slower in the evening, and the migrating motor complex (think of it as the road sweep after a car accident, clearing the small intestine between meals) is slower at night too! Not only that but the liver seems to have its own circadian regulation to synchronise with periods of feeding and fasting. Findings here Factors which inform circadian rhythm: Light Mealtimes Sleep Exercise Temperature If your pet has recently experienced changes to their mealtimes, walk times, or had disrupted sleep, this could play a role in their changing bowel habits. Dehydratio This is particularly relevant to cats. They historically have a low thirst drive and sadly, chronic kidney disease is a risk factor for constipation (diagnosis is increasing year on year). As we noted earlier, the large intestine reabsorbs fluid, and it is here that generally dictates how solid the poop is likely to be. The body always wants to maintain homeostasis, so it will always try to balance its levels. For the cat who isn’t gaining much moisture from their diet, there isn’t much water to reabsorb in the large intestine, but by gum, it will try! This is why we always advocate a fresh food diet for your pet. Always have fresh, filtered water available too. A worthwhile read on cats, hydration and kidney disease here. Obstruction Obstruction can be as sinister as it sounds, blockages caused by foreign objects like toys, plastic bags or elastic bands, but it can also be a medical obstruction caused by a tumour, abscess or granuloma. That said, as pets regularly groom themselves, they
Here at My Pet Nutritionist, we get asked about vaccines a lot. We have plenty of data and scientific, validated research on the core DHP vaccines, but then we have the supposed concern of Leptosporosis, so here is our presentation of the facts. Leptospirosis is a bacterial infection with around 250 serovars. It affects dogs, most mammals and marsupials and even humans. Leptospirosis can be found worldwide but is most prevalent in warm humid countries that experience heavy rain fall. The bacteria can be found in puddles, stagnant water and moist soil that has been infected by the urine of an infected animal. Leptospirosis is transmitted by drinking infected water, through open wounds and through mucus membranes like the eyes and nose. Symptoms can range from mild, subclinical infection with little to no symptoms at all, to multi-organ failure and in some cases death. Signs of leptospirosis are fever, muscle tenderness, vomiting, diarrhoea and jaundice to liver and kidney failure. In 80-90% of cases, leptospirosis is treatable with doxycycline. Low Phosphorus Beef {br} The leptospirosis vaccine is made by Nobivac MSD animal health, a subsidiary of Merck. There are two vaccines available, Nobivac Lepto 2 which covers two serovars and Nobivac Lepto 4, that covers four serovars. We must note that the L3 serovars is not found in the U.K., so one quarter of the vaccine is totally unnecessary. Vaccinating dose not prevent your dog from getting leptospirosis, it is only meant to lessen the severity of the illness. Interestingly a post vaccination survey done by Canine health concern, observed and analysed the history of over 3,800 dogs post vaccination, found that every single dog in the study with leptospirosis, contracted it within three months of the vaccination, which begs the question are we reducing the severity or actually making our dogs more susceptible to it!? Findings here In a 2007 study completed before the availability of the Lepto 4 vaccination, 1,241 healthy dogs, some of whom had been vaccinated and some had not, found that 24.9% of dogs had antibodies 1-6 different serovars. This shows these dogs had natural exposure to these serovars and had built some natural immunity to them. Findings here In cases of the vaccinated dogs, some showed antibodies up to 1,745 days after vaccination. Is this from the vaccination? If so, why are we annually vaccinating? If not, is this from natural exposure? If so, why is it necessary to vaccinate? In 2013, Christopher Ball from Liverpool university wrote his thesis on leptospirosis. This was funded by MSD the manufacturer of Nobivac leptospirosis vaccine. A questionnaire was sent out to 472 vet clinics all around the U.K.. They were asked how many cases of leptospirosis had been seen in the last 12 month’s. 89 clinic’s returned the the questionnaire, the majority reported no cases at all in the last twelve months, only 13 of the practises reported a case within the last twelve months, of which only 3 had been confirmed by laboratory test. None of the practises in the study reported suspecting or confirming two or more cases of leptospirosis in the previous 12 months to the study. With these figures, we need to ask ourselves how prevalent leptospirosis is in the U.K.? And does it really warrant the risks associated with the vaccine? Gut Guardian We may need to look to other means for evidence of adverse reactions, there has been a Facebook group set up called “Novibac Lepto 4 -our experiences” with 35,000 members and growing. Some of whom are professionals all sharing their experiences of this vaccine. There are some extremely harrowing and heartbreaking stories in the this Facebook group. The manufacturer’s of the vaccine and some professionals will say these stories are anecdotal, this may be so, but it reaches a point when ‘coincidence’ can’t be ignored when so many of the same stories are being shared. The World Small Animal Veterinary Association (WSAVA) say that leptospirosis is not a core vaccine and in 2013 Christopher Day said in his thesis due to the perceived low rates of infection in the U.K., the leptospirosis vaccine is not considered a core vaccine, yet recently the BSAVA (British Small Animal Veterinary Association) made it a core vaccination. We would like to know what research and data this is based off. Leptospirosis is a killed vaccine (not live) and therefore contains adjuvants such as mercury and aluminium hydroxide, to stimulate the immune response when the vaccine is administered. This may potentially make it more dangerous in terms of adverse immune events in some dogs. Adverse reactions can be instant or within 7 days of administration. Symptoms can be anything from, sickness, diarrhoea, welts and swelling at the injection site, swollen glands, hives to anaphylactic shock, seizures, auto immune responses and even death. Vaccinosis however, is a disease resulting from a vaccination. This can occur many weeks and even months after the vaccine has been administered, hence why it can be difficult to get a vet to acknowledge and report adverse reactions. Leptospirosis is a zooatonic infection which means we can contract it from other animals. Lepto is known as weils disease in humans and we are often led to believe (from our vets) that it’s very important to vaccinate our dogs due to the risk of leptospirosis to ourselves and our families. However, on the NHS website it say ‘it’s very rare” to contract leptospirosis from our pets. When we take into consideration the low number of cases of leptospirosis, in Christopher Days study, and the fact the NHS have said its very rare to contract it off our pets, this would defiantly go against the consideration of this vaccination for our dogs. Findings here Health and safety executive .gov.uk say there is are around 50 cases of weils (Lepto) disease in the U.K. per year, out of a population of 65 million people. Some of those cases are brought back from abroad, its treated with antibiotics and there is no
Whether you describe your dog as a drama queen or not, most dogs are incredibly good at hiding pain. This mechanism makes complete evolutionary sense. Showing pain, showed a weakness of which predators could take advantage of. Here at my Pet Nutritionist, when you book for a consultation, we ask certain questions to determine if undetected pain could be present, amongst other things. Whilst you may immediately consider the dog limping or struggling to get out of their bed, chances are by this point, the pain has moved past a threshold that they are unable to hide. Our best chance as owners is to notice the subtleties of pain in the canine and know when to seek further help. So let’s explore the mechanism of pain in a little more detail, signs to watch out for what we can do for our four-legged friends. What is pain? We’ve all stubbed our toe in the middle of the night or stood on a piece of lego. This is acute pain, the event, despite being painful, is in fact short lived. This can occur for our dogs too, when they run into a plant pot in the garden for example. But pain can become chronic and this is often what they hide. Chronic pain is more commonly associated with musculoskeletal issues in our dogs, in cases of dysplasia or arthritis. But it can also be associated with gut dysfunction and many other diseases in the body. The origin of all pain is inflammation. Irrespective of the type of pain whether it is acute or chronic pain, peripheral or central pain, nociceptive or neuropathic pain, the underlying origin is inflammation and the inflammatory response. So what is the inflammatory response? Inflammation is a normal response of the body to protect tissues from infection, injury or disease. The inflammatory response begins with the production and release of chemical agents by cells in the infected, injured or diseased tissue. These agents cause redness, swelling, pain, heat and loss of function. Inflamed tissues generate additional signals that recruit leukocytes to the site of inflammation. Leukocytes destroy any infective or injurious agent and remove cellular debris from damaged tissue. This inflammatory response usually promotes healing but, if uncontrolled, may become harmful. Acute inflammation typically lasts only a few days. If a wound gets hot, turns red, hurts, and swells, we recognise that inflammation is at work. In this instance, inflammation is a beneficial process, serving to immobilise the area of injury as the rest of the immune system mobilises to heal. The treatment of acute inflammation, the administration of non-steroidal anti-inflammatory agents, provides relief of pain and fever. In contrast, chronic inflammation lasts weeks, months or even indefinitely and causes tissue damage. In chronic inflammation, the inflammation becomes the problem rather than the solution to infection, injury or disease. Chronically inflamed tissues continue to generate signals that request help from leukocytes in the bloodstream. When leukocytes reach the tissue, they bring inflammation to the party. This chronic inflammatory response can break down healthy tissue in a misdirected attempt at repair and healing. As mentioned, perhaps the most understood cause of pain is joint inflammation. Repeated trauma or stress to the joint, incurred during everyday use, training, or performance, is often the cause of joint inflammation. You will notice the familiar symptoms like lameness, swelling and heat. The initial inflammation usually involves only the soft tissue structures of the joint, and cartilage damage is often not present at this stage. This inflammation allows leukocytes, or white blood cells, which are normally filtered out of the joint, to invade the joint space. The inflamed synovial membrane and the leukocytes release destructive enzymes such as free radicals, cytokines, and prostaglandins, all of which are potentially damaging to the cartilage. This is primarily what anti-inflammatory medications work on, they generally inhibit the production of prostaglandins. Joint degradation is an unfortunate side effect of ageing, but this can occur a lot sooner in those working or particularly active dogs. Degradation can also occur as aside effect in cases of dysplasia; as the bones are not sitting in the joint correctly, friction results in excessive degradation and subsequent inflammation and pain. In these cases, treatment is more relating to pain management and making your dog more comfortable. There are a number of practitioners that specialise in musculoskeletal concerns in the canine. Specialist Vets Chiropractors Vets Qualified in Acupuncture Canine Massage Therapists Hydrotherapists Physiotherapist Whilst there are breeds that are genetically predisposed to conditions like hip or elbow dysplasia, being mindful of appropriate nutrition during development along with appropriate exercise during growth is essential. Large breeds undergo a rapid stage of development and so essential nutrients to bone mineralization should be included in the diet; namely whole food sources of calcium and phosphorus along with vitamin D. Weight should also be monitored in the growing puppy. The bottom line? Count the nutrient dense, fresh food additions to your puppy’s diet, not the calories. As an aside, there is a lot of attention paid to the calcium: phosphorus ratio in the growing large breed. The note to make is that organic sources of both minerals are slowly and less efficiently absorbed; so, in fresh fed dogs this is less of a concern in terms of excess. Phosphorus found in meat products however is more efficiently absorbed than that found in plants. Findings here Gut Pain As humans, we are familiar with tummy aches, well, this can also biochemically occur in the dog as well. They just can’t tell us about it. As we have noted, pain is inflammation, and there are certainly plenty of things that can result in inflammation. Inflammation in the gut can be a result of things getting to places they shouldn’t really be, and so the immune response is summoned. Potentially harmful substances can find their way into the gut, but anything that is identified as non-self, kicks up the immune system. This can simply be a poorly digested protein. If
Whether you are a seasoned raw feeder, rather new to the journey, the Gordon Ramsey of the home-cooked dog food world, or simply want in to add some fresh food to your commercial pet food, here at My Pet Nutritionist, we have a few favourite fresh food additions. Try saying that on a Friday night! Anyway, they all pack a fair punch in terms of nutritional value, so we’ve decided to let you in on our secrets. 1) Green Leafy Vegetables! Top of the list for a reason. Green leafy vegetables include spinach, kale, watercress and broccoli. In green leafy vegetables you will find vitamins A, C, E and K along with many of the B-vitamins. These vegetables also contain carotenoids. Carotenoids act as an antioxidant, deactivating free-radicals and limiting the damage they can cause. There are two broad classifications of carotenoids: carotenes and xanthophylls. The latter containing oxygen, whilst the former do not. Xanthophylls include lutein and zeaxanthin, which are both primarily associated with eye health. They are often indicated in cases of macular degeneration. Beta-carotene one of the carotenes is turned into Vitamin A and is found in those green leafy veg like spinach and kale. Vitamin A is important in maintaining healthy skin, mucous membranes and supporting a functioning immune system. Vitamin A is regularly implicated in abnormal hair cycling, both deficiencies and toxicities can cause issues. For that reason, it is generally deemed that a whole food diet is more than capable of providing healthy levels. Green leafy vegetables also contain a rich source of folate, this is after all, where the name came from. Folate comes from the Latin folium, because it was first found in leaves! Folate functions as a coenzyme in many processes in the body. It is needed to make DNA and other genetic material and is essential in cell division. In short, it helps tissues grow and makes cells work. Folate is also involved in neurotransmitter synthesis, so it is implicated in mood and subsequently behaviour. And, one particular study showed a reduced rate of bladder cancer when leafy veg was added to the dog’s diet! Findings here So, lightly steam your kale or spinach, or blitz it up in a blender, pop it in a freezer mould and add them to your dog’s bowl! 2) Mushrooms! There are literally thousands of species of mushrooms on the planet, so it can be a challenge to know where to start. But they are incredible in terms of the benefits they can provide. Mushrooms can be involved in the prevention of certain disease, the regeneration of damaged cells, the protection of tissues and cells and used during treatment of existing conditions. The bioactive compounds of mushrooms include polysaccharides, proteins, fats, ash, glycosides, alkaloids, volatile oils, tocopherols, phenolics, flavonoids, carotenoids, folates, ascorbic acid enzymes, and organic acids. Reishi mushrooms are adored for their immunomodulating benefits. In short,they have regularly been used successfully in chronic autoimmunity and allergy cases. Maiitike mushrooms have been used for their antibacterial function and cordyceps have been seen to inhibit inflammatory responses throughout the body. Findings here Certain species have been seen to stimulate natural killer cell function, supporting the innate immune system in the body. Mushrooms are widely accepted a having anti-cancer properties. The bottom line? Mushrooms have been seen to be: Anti-inflammatory Anti-microbial Antioxidant Prebiotic Anti-diabetic If you are looking to add mushrooms to the bowl, cook them fully, lightly sauteed is fine. If you are looking to use them therapeutically, then please feel free to get in touch for advice and guidance. 3) Eggs! There’s a reason eggs are the protein reference. They contain the full essential amino acid profile. Eggs contain essential lipids, proteins, vitamins, minerals and trace elements including vitamin A, iron, vitamin B12, riboflavin, choline, zinc and calcium. Egg proteins are distributed equally between egg white and egg yolk, whilst lipids, vitamins and minerals are essentially concentrated in the egg yolk. The yolk is a rich source of linoleic acid and as we know, LA is abundant in the epidermis of the skin, so eggs are a great source of food to support skin health. The one thing that eggs are rarely associated with is being anti-microbial. But there are a range of proteins found in the egg that exhibit antibacterial, antiviral, anti-fungal and anti-parasitic activity. Avidin is seen to target bacteria, lysozymeis seen to target bacteria, virus and fungi, and cystatin targets the above with parasites included! Chicken eggs also contain a range of antioxidant compounds that include vitamins, carotenoids, minerals and trace elements. They have demonstrated reductions in pro-inflammatory cytokines in vitro. Conclusions suggest the supplementation of egg yolk proteins to reduce intestinal oxidative stress. Findings here Lysozyme, an enzyme found in eggs has been indicated in the treatment of inflammatory bowel disease; it has been seen to protect in cases of colitis. Largely from its anti-inflammatory properties. Findings here Where you source your eggs from is essential, free-range birds produce more nutrient dense eggs than caged. Free-rearing results in much higher levels of tocopherol, alpha-tocopherol and lutein content. The anti-microbial function of the egg white is also modulated when hens are exposed to environmental microbes. Findings here So, if you want to add egg to the bowl,source free-range and organic. You can feed raw or cooked, and the egg shell is perfectly safe to feed too! 4) Berries! If you’re in the middle of a yeast or gut healing protocol, then berries may not be the first thing to add to your bowl, but for the typical dog, they are a beautiful addition. Berries contain many vitamins but that also have an impressive antioxidant profile. Anthocyanins (Greek anthos =flower and kyáneos = blue) belong to the flavonoid group of polyphenols, which are responsible for the red and blue colour of plant organs such as fruits, flowers, and leaves. Many studies have linked these compounds with antioxidant, anti‐inflammatory and anti-carcinogenic properties, protection against both heart disease and cancer, as well as a
Hyperthyroidism is commonly accepted as the most common feline endocrine disorder. These are the words found in a Journal of Feline Medicine and Surgery. Here at My Pet Nutritionist, this worries us for two reasons. 1) That hyperthyroidism is such a common disorder 2) That it is accepted Being noted as an epidemic, we think it deserves a little attention. So, let’s explore hyperthyroidism in a little more detail, and what, if anything we can do about it. Until the late 1970’s, there appear few references to hyperthyroidism in the cat. Were there sudden developments in veterinary science, or was something else afoot? What is the thyroid? The thyroid gland is part of the endocrine system. It is a small butterfly shaped gland in the neck. One of the main functions is to produce hormones to regulate the body’s metabolism (the process that turns food into energy). These are more commonly referenced as T3 and T4. Hyperthyroidism is when the thyroid is hyperactive – there is an increased production of thyroid hormones. Symptoms of Hyperthyroidism: Weight loss Increased appetite Increased thirst Increased activity Increased heart rate Poor coat condition Mild/moderate diarrhoea (yet some cats can demonstrate generalised weakness lethargy and loss of appetite) Many cases of hyperthyroidism can be medicated, and so complications are rare, but they can include: Cardiovascular changes High blood pressure Kidney damage (more commonly asa result of treating hyperthyroidism) Diagnosis is usually based on enlargement of the thyroid glands, but this is not always visible. A blood test will confirm levels of thyroid hormones. Treatment is generally with thioamides, which in short act as anti-thyroid medication. They do not cure, simply control, but they must be administered daily. Side effects of this medication can include poor appetite, vomiting and lethargy. Although in some cases, the thyroid gland has been removed. So, what causes hyperthyroidism? So far, there are two main categories of risk factors for hyperthyroidism in the cat. – Nutritional deficiencies – Exposure to thyroid-disrupting compounds There are too genetic risk factors associated with hyperthyroidism, for example Siamese or Persian cats are less likely to suffer with the condition where as domestic shorthaired are the more commonly affected. Findings here Nutritional Deficiencies As claimed in the Journal of Feline Medicine and Surgery, “the feeding of commercially prepared cat food is one leading candidate as a major risk factor for the development of thyroid pathology and hyperthyroidism in cats.” It claims that all epidemiological studies to date have identified feeding an increased proportion of commercially prepared cat food is a risk factor. There are arguments that in the last few decades, the cat food industry has been on a bit of a rollercoaster with iodine. First, they considered high iodine was implicated in hyperthyroidism, so they significantly dropped levels, and since, iodine content has ranged from non-detectable to extremely high, depending on the manufacturer. Iodine is a key element in the synthesis of thyroid hormones. Low iodine results in low thyroid hormone,which spurs the pituitary gland to increase its secretion of thyroid stimulating hormone (we all want to survive after all). Persisting high TSH leads to enlarged thyroid glands. Interestingly, when studied, cats fed a commercially prepared diet who were relatively deficient in iodine, were four times more likely to develop hyperthyroidism than cats supplemented with iodine. Findings here Simple iodine supplementation is not the answer to the question, as is it considered merely a co-factor in a range of causes. Others include, exposure to thyroid-disrupting compounds in the environment, drinking water or diet. Interest in this first appeared with the introduction of fire-retardant chemicals in many household items. The rates of hyperthyroidism seemingly increased with the introduction of these products. Findings here Cats are hyper-carnivorous and have lower activity of certain enzymes involved in both phase I and phase II detoxification pathways. These phases are responsible for breaking down toxic substances in the body (both internally produced and externally exposed). This therefore limits their ability to metabolise toxins. This poor metabolism of xenobiotics is thought to alter the structure and function of the thyroid gland, not only that but there are considerations that it too effects the elimination of T4 hormone, of which leads to high circulating levels. There is evidence which correlates high levels of toxic compounds with feline hyperthyroidism, these compounds are also higher than found in humans that the cats live with (suggesting issues in metabolism). There is a risk associated with commercial food that contain isoflavones and phthalates coming from soy and corn. Both of these compounds are known as endocrine disrupters. These vegetable substrates are detoxified by a particular pathway which is known to be slow in cats. Findings here Being an indoor cat is seen as a risk factor because of the exposure to persistent organic pollutants. Dust is a matrix collecting a range of volatile chemicals released from indoor products like flooring, textiles, furniture, and of course human skin (containing beauty or cleaning products). The heightened exposure is thought to be due to cat grooming behaviour – licking the dust off their coats and ingesting it. Findings here Certain flame-retardant compounds have also been found in commercial cat foods, and data has suggested that the cat liver struggles to metabolise these compounds. These PBDEs (polybrominated diphenyl ethers) contain bromine atoms which mimic iodine and so they may interact with thyroid binding proteins. Cats with hyperthyroidism have also demonstrated higher levels of PFAS which are polyfluoroalkyl substances which are found in products with grease repelling functions, like surface sprays, and floor polishes. Findings here Interestingly, there is a correlation between litter use and hyperthyroidism in cats. Those that use litter boxes more often, are more likely to receive a diagnosis. That along with receiving treatment for ectoparasites. Findings here BPA exposure has also been linked to hyperthyroidism in cats. As we know BPA is found in food, air and soil! Again, cats have a slower pathway in eliminating it. BPA binds to thyroid receptors, competitively displacing hormones. So, what can we do
Any quick search on the internet will populate a range of breeds that are seemingly notorious for being fussy eaters. If you have a basenji, husky or yorkie, it looks like you’re signed up for a lifetime of stressful meals. Except here at My Pet Nutritionist, we don’t believe everything we read on the internet. Whilst all those breeds could indeed be fussy eaters, so can many more. And they are. It is perhaps one of the more common questions we are asked, “how can I get my dog to eat?” Being a fussy eater can be technically defined as an eating disorder, and there are a number of causes. From behavioural to biochemical, let’s take a look at the complex world of the fussy eater. The Function of Eating Food components are the main sources of energy for the canine body. Not only that, but it provides the compounds needed for each cell to do its job. As the body carries out its tasks, it uses fuel and compounds, as reserves run low, signals bounce around the body to kickstart feeding behaviour. This is hunger, a physical need to eat. Appetite is quite simply the desire to eat. Hunger and appetite can be at odds. You may want to eat, but not need to, and you may need to eat, but not want to (in times of stress for example). Appetite and hunger are largely controlled by the brain and a range of hormones. The Brain In the brain sits the hypothalamus. Through its connection to the pituitary gland, it modulates the endocrine system. It is involved in a range of daily activities including temperature regulation and energy maintenance. We know it plays a role in eating behaviour as several lesions to small areas of it can result in overeating and under eating. The lateral hypothalamus is defined as the feeding centre and the ventromedial hypothalamus is defined as the satiety centre. This is largely an oversimplification, but it certainly demonstrates the role. The hypothalamus receives information from the digestive system like stomach extension, chemical nature of ingested food and the metabolic activity of the liver and uses it to maintain energy balance. It also receives information from the emotion/reward system. Food is a rewarding object that induces pleasant emotions. Findings here The amygdala is largely responsible for this. Studies have demonstrated that when the reward value of food decreases, so too does eating motivation. Sadly, these studies often include the injection of lithium after eating, of which causes discomfort. But it does raise an interesting point in terms of the fussy eater in your life. We’ll revisit this later. Food reward is elicited by several events that occur before it even passes through the oesophagus, namingly the appearance and shape of the food, the taste and smell and then the pleasure of swallowing the food. We know this because in tube-feeding studies, reward sensations are reduced. In short, when subjects were no longer allowed to taste or chew it, they did not want to eat it. That said, in sham studies, when animals are denied nutrition because everything swallowed leaks out of a tube connected to the oesophagus, they eat and swallow more than usual, but they are still unsatiated. This tells us just how complex eating behaviour actually is. And provides food for thought for the gluttonous dog (on the other side of the scale). Hormones Hormones are probably the most talked about in terms of eating behaviour. You’ve all likely come across leptin and ghrelin. Leptin is produced in adipose cells, or fat cells. So, the more fat cells there are,the more leptin. In short, the more fat available in reserves, the less you need to eat. If you have no fat cells, you need to conserve your energy until you next find food. Leptin crosses the blood-brain barrier, and there are high numbers of leptin receptors found in the hypothalamus, brain stem and other regions of the brain. Rising leptin in a fed state inhibits food intake by suppressing a range of peptides involved in eating behaviour. Ghrelin is predominantly secreted in the stomach and it too modulates cells found in the hypothalamus by increasing excitatory inputs and decreasing inhibitory inputs. Here we are talking about neurotransmitters. These chemical messengers modulate much of our and our pet’s behaviour and they either make something do something or stop something from doing something. Whilst dopamine can be both inhibitory and excitatory, ghrelin is seen to have a large influence on the release of dopamine via increases in cell excitability. As dopamine is involved in reward and motivation, ghrelin is thought to target the motivational functions geared to gaining food and to select those which are more rewarding (high calorie). Findings here In eating disorders, dopamine is one of the neurotransmitters that gets a lot of attention. In times of reduced food intake (fussy eating), dopamine neurons are activated, in the short-term rewarding the lack of food. It is considered that it is a physiological response in an attempt to increase motivation to forage for food. Findings here However, there are also other mechanisms in which the dopaminergic system comes into play for the fussy eater. A central feature of the dopamine neuron response is that it is triggered by unexpectancy. After receiving an unexpected reward like food (or how many likes our recent post has got on social media) a dopamine surge is elicited. When this becomes a regular occurrence, the dopamine signal is triggered by the conditioned stimulus in predicting the reward. However, the dopamine system does not respond when the reward is received. If the reward is predicted, then not received, there is a dip in dopamine activity. What this means, is that your dog may do the song and dance ready for their bowl of food, but then walk a way as soon as it is placed in front of them. The reward they predicted (tasty food), isn’t what was received. The other neurotransmitter that gets a
As we look at photos or videos of dogs devouring their food, when we are dealing with a fussy eater, the question we always sit with is; why won’t my dog do that? Being a fussy eater can be technically defined as an eating disorder, and there are a number of causes. From behavioural to biochemical, let’s take a look at the complex world of the fussy eater and we’ll share our top 5 tips for dealing with them. Eating Behaviour Appetite and hunger are largely controlled by the brain and a range of hormones. In the brain the hypothalamus receives information from the digestive system like stomach extension, chemical nature of ingested food and the metabolic activity of the liver and uses it to maintain energy balance. It also receives information from the emotion/reward system. Food is a rewarding object that induces pleasant emotions. Studies have demonstrated that when the reward value of food decreases, so too does eating motivation. Sadly, these studies often include the injection of lithium after eating, which causes discomfort, but it does give us food for thought, pun intended, if our dog is eating food which may be causing them discomfort. The most discussed hormones when we are looking at appetite and hunger are leptin and ghrelin. Leptin is produced in adipose cells, or fat cells. So, the more fat cells there are, the more leptin is produced. In short, the more fat that is available in reserves, the less you need to eat. Ghrelin is predominantly secreted in the stomach, and it too modulates cells found in the hypothalamus by increasing excitatory inputs and decreasing inhibitory inputs. Here we are talking about neurotransmitters. Ghrelin is seen to have a large influence on the release of dopamine via increases in cell excitability. As dopamine is involved in reward and motivation, ghrelin is thought to target the motivational functions geared to gaining food and to select those which are more rewarding (high calorie). However, there are also other mechanisms in which the dopaminergic system comes into play for the fussy eater. A central feature of the dopamine neuron response is that it is triggered by unexpectancy. After receiving an unexpected reward like food (or how many likes our recent post has got on social media) a dopamine surge is elicited. For the dog who ends up with the same bowl of food every day, there is no longer unexpectancy. The other neurotransmitter that gets a little attention in terms of eating behaviour is serotonin. Serotonin has a well confirmed role in the regulation of eating behaviour. Serotonin is a key player in feelings of nausea – so higher levels could be produced in response to something not sitting quite right in the digestive system. What’s super interesting is that serotonin is metabolised largely by monoamine oxidase (MAO) so it stands to reason that MAO inhibitors can contribute to high levels in the body. Sadly, certain insecticides found on flea and tick collars contain MAO inhibitors. We must also consider the cascade of hormones that are released during the stress response. Stress is a redirection of resources, and this includes away from digestive function. If our dogs are exposed to acute or chronic stress, it could remove their desire to eat. That said, as an owner managing a fussy eater, the owner too can become stressed and anxious that the dog isn’t getting the nutrients they need to thrive. Unfortunately for us, dogs have incredible olfactory capabilities, meaning they can smell our stress! They also have this capacity to mirror our emotions. And so, when they don’t eat, we get stressed, so they get stressed and may be even less likely to eat. So what does all of this mean? If you have investigated further and there is no underlying health issue that could be causing your dog to be a fussy eater, they could: Be suffering discomfort from the food they are eating, whether this is a sensitivity, or issues with digestion. They are then less likely to want to eat it next time for fear of feeling poorly. Have an undesirable body score – there is a difference between the occasional fussy eater, and the chronic fussy eater. If your dog is only occasionally refusing food, they may well just not need it at that moment. Be bored with their food – this often applies to many commercial foods – dry food we’re looking at you. Having the same bowl of food every mealtime removes the sense of unexpectancy. Be stressed or sense our stress – have there been any recent changes to your routine? Are you more stressed than usual? Has their walking route changed? Have they had an altercation with another dog on a walk? Are there any new pets in the home? Have you moved any furniture? Have you noticed any other changes to your dog’s behaviour? Have there been any changes to your behaviour? Our 5 Top Tips for Managing a Fussy Eater: 1) Establish mealtimes! There’s no unexpected reward if a bowl of food is always laid out. 2) Ditch the dry! What is more boring than the same bowl of pellets every mealtime? Offer fresh food with a range of tastes, aromas, and textures. In addition, variety is the spice of life – use novel proteins – what’s more unexpected than novelty? This can also be helpful if you are concerned your dog may have a sensitivity to certain foods. Finally, cooking alters the aroma and texture of many foods so this can be a great way to entice if needed. 3) Use mealtimes as opportunities to enrich your dog’s life! Offer food in bowls or plates, or even on wooden boards. There is no categoric link between height of feeding and gastric torsion in dogs, so consider at what level you offer their meals; do they need to be raised if they are struggling with any musculoskeletal issues. If they are in pain whilst eating,
Gone are the days when you could walk into a pet shop and have the choice of three types of food: the cheapest, middle of the road and the most expensive. Thank goodness! But we now have a new problem. What on earth do I feed my pet? Do I feed dry kibble? Quite simply, NO. But what about the rest? Canned? Dehydrated? Commercial Cooked? Home Cooked? Commercial Raw or DIY Raw? At My Pet Nutritionist, we can help guide you. In an age of choice, is there a “best diet”? Obviously human research demonstrates fresh feeding over processed foods is far healthier for us and can prevent disease. this applies to our pets too however our job is to look at the pet, holistically and consider all constraints involved. Whilst we know there are rules of thumb for optimal health, much like for us humans, all pets are different. There may be a perfect diet for your pet, but that same diet may not be perfect for your neighbour’s pet. So let’s explore the different foods available to you, what we like about them and what considerations need to be made, if in fact they are your foods of choice. Raw Feeding Raw feeding is just that, when you feed meat, fat, bones etc in their raw state. Within raw feeding there are a number of approaches. You may come across phrases like BARF which stands for biologically appropriate raw feeding, or RMB which stands for raw meaty bones, you may also come across PMR which is prey model raw and more will no doubt appear over the coming years. The basis of a raw diet comes from considering the evolution of the dog. Dogs came from wolves, and before domesticated by humans, would largely fend for themselves; eating whole prey; eyes, brains, guts galore (although there is some data that suggests the guts were often left, which leads to the suggestion that dogs avoid greens when given the opportunity, but this isn’t always the case and some leafy greens pack some punch in terms of nutritional value). The biggest concern around raw feeding is contamination and so, we must practice appropriate handling techniques when handling any raw meat, just read the packaging on your raw chicken fillets. The more recent development is around antimicrobial resistance. Whilst raw food poses a risk, so does the inappropriate use of antibiotics, excessive use of disinfectants and sanitisers, livestock rearing techniques and pesticide use along with many more. Within raw feeding you may come across the 80-10-10 method. This is where you feed 80% meat, 10% bone and 10% offal (organs). This is what most commercial raw food manufacturers offer, in a ground state, as a basis for their meals. But you can also formulate a raw diet to FEDIAF guidelines, again some raw food manufacturers provide products that meet these requirements and offer complete foods. Raw food is in its original state, apart from being processed through a meat grinder in pre-made minces, what you see is what you get. Feeding a raw diet has been associated with a range of improvements and health benefits in pets: Digestibility of crude protein and fat is improved in raw foods compared to dry practical pet foods. Dogs fed a RMB diet have a significantly more diverse microbiome than commercially dry fed dogs. Dogs being fed a RMB diet demonstrated a high digestibility of nutrients. Generally, the size of stool is deemed as a measure of digestibility in pet food manufacturing and in raw fed dogs, stool size significantly diminishes (suggesting improved digestibility). Dogs fed dry kibble compared with a raw food diet have demonstrated altered immune cell gene expression. Keto diets have been seen to improve outcomes in pets with cancer; a raw diet is largely based on keto principles. In a randomised clinical trial in 2012, the positive effect of a raw diet was reported in relation to the formation of calcium oxalate uroliths. Lower calcium excretion was found in the urine of dogs eating a commercial RMBD, compared with excretion in dogs eating a commercial dry extruded diet. An initial analysis of the Dog Risk data (Helsinki University), has revealed a decrease in the incidence of atopy and allergy in dogs that eat RMBDs. It appears that an RMBD fed at a young age has a positive influence on reducing the incidence of atopy in the population. Eating raw meat, raw bone and cartilage gave the strongest association of any the food items consumed in the study. Results have indicated that raw food, fed at a young age, could protect German shepherd dogs from CHD (Canine Hip Dysplasia). All findings here Dogs fed raw food often have less plaque build up and white teeth on raw food. Pets have a dietary requirement for protein and fat. Protein to build and repair cells, fat to provide energy, insulation and to act as signalling molecules. They also have a requirement for a range of micronutrients, those vitamins, and minerals. Meat, bones and offal are full of available nutrients, in their original state. Raw is perfectly able to provide nutrients for your dog to thrive, but the dietary plan may take some tweaking to get it right. Some dogs take to bone straightaway, some need a little longer. Some can tolerate 10% offal, some start to get black, tarry poops without an offal free meal or day. The Poop Gives you the Scoop As much as we’d like to tell you the eyes are the window into the soul; when you’re feeding a pet, if you want to know what’s going on inside, keep a watchful eye on their poop. Too much bone in a raw food diet starts to give white, crumbly poop. Too much offal is usually a black and tarry poop. There are other indicators too, but here at My Pet Nutritionist, we’re not averse to a poop pic, so if you are worried about your dog then please feel free
If you’ve just rinsed your dog off after a meander through the woods, then their licking is likely grooming behaviour. But, here at My Pet Nutritionist, we often see dogs who excessively lick causing abrasions and hair loss. Whilst it’s often indicative of an irritation, there are in fact some other reasons why your dog may start licking in the absence of a jaunt through a muddy puddle. Let’s take a look at some of the more common causes. 1) Stress Yes – stress can take the blame for this one too! Stress is like the wonder woman of life – is there anything she can’t do? For anyone who has spent time investigating dog behaviour, you’ll know that lip licking in particular is a sign of stress. An interesting theory here suggests that because stress depletes essential nutrient resources, lip licking is in fact a sign of malnutrition. Deficiencies in a range of nutrients like vitamin B2, B3, B12, iron, and Vitamins A, C and K and Zinc have all been known to cause irritation and inflammation in and around the mouth and lips. The licking is thought to be an attempt to soothe it. Findings here However, other suggestions explore the idea that the lip licking mechanism is thought to soothe the stress response by activating the parasympathetic nervous system. Yet, this mechanism can apply to licking of all body parts, not just the lips. The stress response is dominated by the sympathetic nervous system; it increases heart rate, widens eyes in order to assess the threat better and redirects resources in order to deal with it. Once the threat has passed, the parasympathetic nervous system kicks in and re-balances. The chief of the parasympathetic nervous system is the vagus nerve, and chewing, along with licking is considered to activate it. Through the action, attention is redirected and focussed on that particular task, not what is making them stressed. Findings here Licking is also deemed a self-soothing behaviour through its association with oxytocin. Oxytocin is a hypothalamic nonapeptide linked to increased levels of social interaction, well-being and anti-stress effects. Oxytocin is released by sensory stimulation (touch) and promotes the release of dopamine whilst decreasing the cascade of stress response mechanisms throughout the brain and body. Oxytocin also modulates the activity of serotonin which as we know is a key hormone in mood stability, feelings of well-being and reward. An element of this could have also been learned behaviour from their mother. Licking and grooming behaviour is a key feature in determining neural changes and fear responses in offspring. Those from low licking mothers tend to show a greater stress-response and proceeded to be low-lickers themselves (if they were female and had their own litter). The same also occurred for high lickers. Findings here The bottom line? Excessive licking could be a response to stress faced by your dog. Watch out for other signs of stress like: Panting in the absence of exercise Salivating in the absence of food Pacing Inappropriate toiletting Unusual vocalising Out of character behaviour Wide eyes Flat ears Abnormal tail placement Work to remove the trigger and support them with other stress reduction strategies. Also, be mindful that chronic stress can deplete essential nutrient resources. our Calm Complex can help support the above signs of stress. 2) Pain Pain is an uncomfortable feeling that gives us and our dogs an indication that something isn’t as it should. Not only are many breeds incredible at hiding pain (it wouldn’t have helped them in evolutionary terms), but they also lack the ability to converse. Whilst we would all love to think we have this form of communication with our dogs, we can miss things, especially if they are pretty crafty at hiding them! The sensation of pain is a necessary function that warns the body of potential or actual injury. Whilst we may think that dull ache in our lower backs is merely an inconvenience, it’s a rather reliable indication that our office chair possibly doesn’t suit us. Pain occurs when a nociceptor fibre detects a painful stimulus on the skin or in an internal organ. This detection is picked up by other receptors in the spinal cord and brainstem and then transmitted to the various areas of the brain. This is done through the incredible work of neurotransmitters. There exists both inhibitory and excitatory neurotransmitters. Those that make things happen, and those that apply the brakes. Inhibitory neurotransmitters are involved in the modulation of pain; including enkephalins and endorphins, serotonin, noradrenalin, GABA, acetylcholine and oxytocin. Excitatory neurotransmitters acting without an inhibiting system results in pain. Which is why pain killers primarily prevent the release of some excitatory neurotransmitters (and can in fact make you or your dog drowsy). When we look at the neurotransmitters involved, it’s possible to see why our dogs may start licking an area that is painful to them. Through touch they can stimulate the release of oxytocin (a modulator of pain). Not only that but self-trauma (excessive licking) is thought to promote the release of endorphins, which are also pain modulators. Findings here This demonstrates how dogs can get into a vicious cycle, they want to lick to soothe, but the more they lick, the more trauma they cause, resulting in more licking. There is also the possibility, that what could have started as a pain response, could turn into irritation, or what started as irritation, could turn into pain and continue the cycle. 3) Itch Itch is a sensation felt on the skin, which causes the desire to scratch. Although initially it could be as simply as scratching something to remove it, itching can become stressful when excessive. When tissues are stimulated by allergens, histamine is released from mast cells. Histamine makes the blood vessels expand, driving blood to the area creating that common swelling among other responses. Specialised nerve fibres are stimulated; when these are processed, the scratching or rubbing reflex is stimulated. The main mediators for the itch
Help! My dog has a loose stool! Well, you wouldn’t be a true dog owner if you didn’t spend time assessing, talking or even sharing photos about your dog’s bowel movements. Here at My Pet Nutritionist, we’re not averse to a poop pic (we get quite a few in a day), but there are some common reasons why your dog may develop high scores on the Bristol Stool Chart. Technically that’s a chart for humans, but we can learn similar lessons. Here is our Ultimate Dog Stool Guide on colour and consistency. So, let us take a look at some of causes of runny poops in dogs. 1) Infection It goes without saying that a nasty gastro bug can result in runny stools. It can be all sorts of different colours, but more often that not it can be described as like turning a tap on! Infections can be a result of bacteria, parasites or virus and you may notice other symptoms like lethargy, vomiting or abdominal pain. In some cases, due to the irritation in the gastrointestinal tract, you may find specks of blood in the stool too. If nothing sinister, a fast followed by a bland diet will start to show improvements, but if there are other clinical signs or no improvements then its always best to get your dog checked out by your Vet. A vet call is always warranted for young puppies or elderly dogs. 2) Gut Dysbiosis Stool consistency strongly correlates with all known major microbiome markers. What this means is good poops are a sign of a healthy gut. But you knew that! Gut dysbiosis is when there is an imbalance between the good and bad bugs found in the gut. This can be for a number of reasons: Antibiotic use Steroid use Diet diversity Environment – exposure to toxins Birth method Use of flea and wormer treatments Vaccination When we are looking to heal the gut, we follow the 4 R’s. Remove (food antigens that could cause reactivity) Repair (the gut lining/endothelial, possibly known as leaky gut) Restore (the good bacteria in the gut) Replenish (with enzymes if necessary to reduce immediate inflammation) Signs of gut dysbiosis will usually manifest as regular sub-optimal bowel movements, if you think they may be an issue faced by your pet, we are more than happy to help! 3) Food Sensitivities/Allergies Allergies are in fact incredibly rare (at around 10%), but sensitivities are one of the most common issues discussed here at My Pet Nutritionist. An allergy is the result of an over-zealous immune system. It’s like going for the theory part of your driver’s test when you are told you are limited with how many clicks you can make on your hazard perception. Well, in cases of an allergy, the immune system didn’t get the limited clicks memo and gets a little trigger happy! The basic function of the immune system is to get rid of potential threats; it can do this by engulfing parasites, and you guessed it, causing a swift exit in the form of diarrhoea. But diarrhoea is also a sign of a food intolerance. Rather than an immune response per se, an intolerance involves the digestive system; in short, it struggles to process a particular food. This can be due to a lack of enzymes to digest a particular food, a sensitivity to chemicals found in certain foods or health issues like irritable bowel syndrome (which is often linked to bacterial overgrowth, stress, post-infection and genetics). In cases of food sensitivities, an elimination diet is the first port of call, to establish the key offender and we again follow the 4R’s in gut healing. 4) Stress Our pet’s stress response is much like ours as a human. Eustress or good stress can improve alertness and motivation and provide a helping hand in performance. But distress is the one we need to worry about, especially if it becomes chronic. Stress is generally known as the fight or flight response. The cascade of changes occurring in the body redistribute resources; blood is directed to the brain and limbs to figure out what to do, and whether to run away or stand and fight. Despite us making evolutionary progress, neither us or our dogs have yet evolved past this response and so even potentially minute stressors result in this redistribution of resources; taking ours and our dog’s ability to effectively digest food with it. Digestive function returns when the parasympathetic system kicks back in; once the stressor has passed (but incases of chronic stress, this doesn’t occur). In times of chronic stress, food is ingested, but not digested properly. This also explains why you may notice your dog has “adrenaline” poops on walks, or when they get particularly excited. As they are doing their zoomies, the body interprets it as stress and so directs resources where they are needed (not to the effective digestion of food). This is short lived and the dog’s next poop that evening has usually returned to its normal presentation but, under long term stress, this lack of digestion demonstrated by recurring loose stools, can result in a range of health issues thanks to the malabsorption of nutrients. If you consider your dog’s loose stools are generally associated with certain stressful events, the key is to remove the trigger where possible. Counter conditioning is often successful in supporting their future stress-responses. Stress does deplete essential nutrients, so opting for a fresh-food diet with excellent bioavailability is vital in supporting your dog during these times. 5) Underlying Malfunction Loose stools are the result of food being passed through the colon too quickly; with less fluid being absorbed by the body, the result being that puddle in your garden. As we mentioned earlier, a lack of digestive enzymes can be a reason for this. The digestive system comprises the GI tract, pancreas, liver, and gallbladder. If there is an underlying malfunction in any of these, you may find abnormal stools. The pancreas
You’re in the middle of your daily grooming session and you notice a thinning patch of hair on your canine companion. Without any obvious recent trauma to result in scar tissue and no replacement hair, your mind starts going to all the places it shouldn’t. A quick google search likely doesn’t help but well here at My Pet Nutritionist, we hope to give you possible solutions. There are in fact a number of reasons why your dog may start losing his hair, or having thinning areas, so let’s look at them in a little more detail. Hair growth occurs in stages: – Anagen – the growing phase – Catagen – the transition phase – Telogen – the resting phase – Exogen – the shedding phase An intact hair coat is maintained by the lifelong cycling of these phases. When a hair follicle has passed the telogen stage and shed it, if it remains empty for a period of time it is deemed kenogen. The anagen phase is restarted when epithelial stem cells are in good form. Stem cell activity is dependent on many complex interactions including: Immune cell function Nerve fibres Hormones Genetics Daylight Nutrition Circadian rhythm And stress 1. Hormones So much of a factor, there is a condition deemed hormonal alopecia in dogs. This can be linked to neutering with many owners reporting hair loss or thinning post neutering. But when we say hormones we are also considering thyroid hormones. The thyroid gland is in fact active in the initiation of hair growth and replacement. Located in the neck near the trachea or windpipe, this gland produces hormones which regulate metabolism. Both hyperthyroidism and hypothyroidism can result in hair loss in the dog although hypothyroidism is likely the more commonly occurring form of hormonal alopecia in dogs. Initially hair loss is patchy, the coat is dry, the hair is brittle and easily pulled out. Quite often hyper pigmentation occurs. In some cases, secondary pyoderma and seborrheic dermatitis may follow. The hair that remains is often stuck in the telogen or resting phase, hence it’s poor condition. Other signs of hypothyroidism includes: Weight gain Lethargy Increased susceptibility to infections Slow heart rate Abnormal nerve functioning which presents as non-painful lameness or lack of coordination Keratoconjunctivitis or dry eye Fat deposits in the corneas of the eyes Thyroid function is easily assessed through screening and if it isn’t running all on cylinders, then it can often be managed. 2. Nutrition It wouldn’t be a My Pet Nutritionist blog without mentioning nutrition now would it. Nutritional status affects hair growth and maintenance. Every cell in every body, including our dog’s, needs basic components to carry out their function. This includes immune cells, stem cells and hair follicles. The root of a hair is made up of protein cells and nourished by nearby blood vessels. As it grows, sebaceous glands near the hair follicles produce sebum which is made up of triglycerides, wax esters and cholesterol. Sebum lubricates the skin and hair to protect it from friction and therefore makes it impervious to moisture and/or pathogens. The hair moves through its phases, but new data is suggesting that even though telogen is defined as the resting phase, hair follicles aren’t doing that much resting. In fact, much cellular activity occurs during this phase so that tissues can regenerate and grow new hair. This means there is a demand for nutrients. Time and time again there has been a link made between nutritional status and hair health and growth. Low vitamin D status has been implicated in cases of alopecia Over-supplementation of Vitamin A is associated with alopecia In a Biotin deficiency signs include hair loss, Folate deficiency can result in hair, skin and nail changes Vitamin C is known to aid iron absorption, the latter being implicated in hair loss Hair loss is a common sign of zinc deficiency Hair loss can be seen in Iodine deficiency, also a mineral that aids thyroid function All things considered we would advocate afresh food diet which includes: Meat protein (haem iron is found in meat, chicken and fish and is more easily absorbed that non-haem iron found in plant foods). Oily fish (source of Vitamin D) Organ meats (good source of biotin) Leafy greens (folate came from the latin folium because it was first found in leaves!) Berries (source of Vitamin C) Meat and shellfish (source of zinc) Be mindful if you are tackling any other health concern which requires the elimination of any of these foods, we can always help you find alternative nutrient sources. 3. Stress Stress, we can confidently say is the bane of everyone’s life, including our pet’s. But what is particularly interesting is that in some cases, hair loss follows months after a traumatic event often making it difficult to connect the dots. As we have mentioned, hair cycles through different phases and all follicles can be at different stages at any onetime. Many will be in the growth phase or anagen phase before hitting telogen and ultimately shedding. But high levels of stress can cause a blanket shift in the phases and bump many of the follicles to telogen, all at the same time. The result? Bald patches or thinning. It makes sense evolutionarily. When facing a threat, what is more important? Brain and muscle function or hair growth? Stress triggers a sort of redistribution of resources, which in the short term is manageable. The issue is when faced with chronic stress. Hair loss as a result of trauma may not appear until the initial stressor has passed, the growth phase has been prematurely stopped, hitting telogen sooner, but then we still have to wait for the shedding to occur. We then need the growth phase to start again, which may or may not happen depending on the recovery from the stressor or in fact whether the host is still experiencing it. Stress also depletes nutritional resources along with impeding the digestion and absorption of them and as
